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@ARTICLE{Hammes:856060,
      author       = {Hammes, Jochen and Drzezga, Alexander and van Eimeren,
                      Thilo},
      title        = {{T}he {R}ole of {T}au {I}maging in {P}arkinsonian
                      {D}isorders},
      journal      = {Current neurology and neuroscience reports},
      volume       = {18},
      number       = {12},
      issn         = {1534-6293},
      address      = {Philadelphia, Pa.},
      publisher    = {Current Science Inc.},
      reportid     = {FZJ-2018-05725},
      pages        = {86},
      year         = {2018},
      abstract     = {Purpose of ReviewDifferential diagnosis of atypical
                      Parkinson syndromes (APS) is difficult as clinical
                      presentations may vary and as there is a strong overlap
                      between disease entities. Aggregations of misfolded and
                      hyperphosphorylated tau proteins are the common denominator
                      of many of these diseases.Recent FindingsSeveral tau
                      targeting positron emission tomography (PET) tracers have
                      been evaluated as possible biomarkers in APS in the recent
                      years. For Parkinson’s disease, dementia with Lewy bodies,
                      progressive supranuclear palsy, and corticobasal
                      degeneration, promising results have been reported with
                      regard to the ability to detect the presence of disease and
                      to discriminate patients from controls. However, the
                      discussion about the specificity of the first-generation
                      radiotracers and their value in the clinical context is
                      ongoing.SummaryA combined interpretation of signal strength
                      and distribution pattern in PET scans with first- and
                      second-generation tracers may be helpful in clinical
                      diagnosis and follow-up of patients with APS},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {572 - (Dys-)function and Plasticity (POF3-572)},
      pid          = {G:(DE-HGF)POF3-572},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:30293094},
      UT           = {WOS:000446537500003},
      doi          = {10.1007/s11910-018-0898-3},
      url          = {https://juser.fz-juelich.de/record/856060},
}