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@ARTICLE{CarballoPacheco:858906,
author = {Carballo-Pacheco, Martín and Ismail, Ahmed E. and Strodel,
Birgit},
title = {{O}n the {A}pplicability of {F}orce {F}ields {T}o {S}tudy
the {A}ggregation of {A}myloidogenic {P}eptides {U}sing
{M}olecular {D}ynamics {S}imulations},
journal = {Journal of chemical theory and computation},
volume = {14},
number = {11},
issn = {1549-9626},
address = {Washington, DC},
reportid = {FZJ-2018-07742},
pages = {6063 - 6075},
year = {2018},
abstract = {Molecular dynamics simulations play an essential role in
understanding biomolecular processes such as protein
aggregation at temporal and spatial resolutions which are
not attainable by experimental methods. For a correct
modeling of protein aggregation, force fields must
accurately represent molecular interactions. Here, we study
the effect of five different force fields on the oligomer
formation of Alzheimer’s Aβ16–22 peptide and two of its
mutants: Aβ16–22(F19V,F20V), which does not form fibrils,
and Aβ16–22(F19L) which forms fibrils faster than the
wild type. We observe that while oligomer formation kinetics
depends strongly on the force field, structural properties,
such as the most relevant protein–protein contacts, are
similar between them. The oligomer formation kinetics
obtained with different force fields differ more from each
other than the kinetics between aggregating and
nonaggregating peptides simulated with a single force field.
We discuss the difficulties in comparing atomistic
simulations of amyloid oligomer formation with experimental
observables.},
cin = {ICS-6},
ddc = {610},
cid = {I:(DE-Juel1)ICS-6-20110106},
pnm = {551 - Functional Macromolecules and Complexes (POF3-551)},
pid = {G:(DE-HGF)POF3-551},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30336669},
UT = {WOS:000450695200053},
doi = {10.1021/acs.jctc.8b00579},
url = {https://juser.fz-juelich.de/record/858906},
}