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@ARTICLE{McMillan:860189,
author = {McMillan, Rebecca and Forsyth, Anna and Campbell, Doug and
Malpas, Gemma and Maxwell, Elizabeth and Dukart, Jürgen and
Hipp, Joerg and Muthukumaraswamy, Suresh},
title = {{T}emporal dynamics of the pharmacological {MRI} response
to subanaesthetic ketamine in healthy volunteers: {A}
simultaneous {EEG}/f{MRI} study},
journal = {Journal of psychopharmacology},
volume = {33},
number = {2},
issn = {1461-7285},
address = {London [u.a.]},
publisher = {Sage},
reportid = {FZJ-2019-00973},
pages = {219-229},
year = {2019},
abstract = {Background:Pharmacological magnetic resonance imaging has
been used to investigate the neural effects of
subanaesthetic ketamine in healthy volunteers. However, the
effect of ketamine has been modelled with a single time
course and without consideration of physiological
noise.Aims:This study aimed to investigate ketamine-induced
alterations in resting neural activity using conventional
pharmacological magnetic resonance imaging analysis
techniques with physiological noise correction, and a novel
analysis utilising simultaneously recorded
electroencephalography data.Methods:Simultaneous
electroencephalography/functional magnetic resonance imaging
and physiological data were collected from 30 healthy male
participants before and during a subanaesthetic intravenous
ketamine infusion.Results:Consistent with previous
literature, we show widespread cortical blood-oxygen-level
dependent signal increases and decreased blood-oxygen-level
dependent signals in the subgenual anterior cingulate cortex
following ketamine. However, the latter effect was
attenuated by the inclusion of motion regressors and
physiological correction in the model. In a novel analysis,
we modelled the pharmacological magnetic resonance imaging
response with the power time series of seven
electroencephalography frequency bands. This showed evidence
for distinct temporal time courses of neural responses to
ketamine. No electroencephalography power time series
correlated with decreased blood-oxygen-level dependent
signal in the subgenual anterior cingulate
cortex.Conclusions:We suggest the decrease in
blood-oxygen-level dependent signals in the subgenual
anterior cingulate cortex typically seen in the literature
is the result of physiological noise, in particular cardiac
pulsatility. Furthermore, modelling the pharmacological
magnetic resonance imaging response with a single temporal
model does not completely capture the full spectrum of
neuronal dynamics. The use of electroencephalography
regressors to model the response can increase confidence
that the pharmacological magnetic resonance imaging is
directly related to underlying neural activity.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {571 - Connectivity and Activity (POF3-571)},
pid = {G:(DE-HGF)POF3-571},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30663520},
UT = {WOS:000458980800007},
doi = {10.1177/0269881118822263},
url = {https://juser.fz-juelich.de/record/860189},
}