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@ARTICLE{Wyss:861573,
author = {Wyss, Christine and Tse, Desmond H Y and Boers, Frank and
Shah, N. J. and Neuner, Irene and Kawohl, Wolfram},
title = {{A}ssociation between {C}ortical {GABA} and {L}oudness
{D}ependence of {A}uditory {E}voked {P}otentials ({LDAEP})
in {H}umans},
journal = {The international journal of neuropsychopharmacology},
volume = {21},
number = {9},
issn = {1469-5111},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {FZJ-2019-02024},
pages = {809 - 813},
year = {2018},
abstract = {Loudness dependence of auditory evoked potentials (LDAEP)
is a widely used EEG-based biomarker for central
serotonergic activity. Serotonin has been shown to be
associated with different psychiatric disorders such as
depression and schizophrenia. Despite its clinical
significance, the underlying neurochemical mechanism of this
promising marker is not fully understood, and further
research is needed to improve its validity. Other
neurotransmitters might have a significant impact on this
measure. Thus, we assessed the inhibitory action through
individual GABA/H20 concentrations and GABA/glutamate ratios
by means of magnetic resonance spectroscopy at 3T in healthy
subjects. The measurements were assessed in the primary
auditory cortex to investigate the association with the
LDAEP, whose generators are mainly in the primary auditory
cortex. For the first time, this study examines the link
between GABAergic neurotransmission and LDAEP, and the data
preliminary show that GABA may not contribute to the
generation of EEG-based LDAEP.},
cin = {INM-4 / INM-11 / JARA-BRAIN},
ddc = {610},
cid = {I:(DE-Juel1)INM-4-20090406 / I:(DE-Juel1)INM-11-20170113 /
$I:(DE-82)080010_20140620$},
pnm = {573 - Neuroimaging (POF3-573)},
pid = {G:(DE-HGF)POF3-573},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29917080},
UT = {WOS:000446178200003},
doi = {10.1093/ijnp/pyy056},
url = {https://juser.fz-juelich.de/record/861573},
}