% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Becker:861914,
author = {Becker, Julia and Mahlke , Nina Sophie and Reckert ,
Alexandra and Eickhoff, Simon and Ritz-Timme, Stefanie},
title = {{A}ge estimation based on different molecular clocks in
several tissues and a multivariate approach: {A}n
explorative study},
journal = {International journal of legal medicine},
volume = {134},
issn = {0044-3433},
address = {Heidelberg},
publisher = {Springer},
reportid = {FZJ-2019-02320},
pages = {721-733},
year = {2020},
note = {Bitte die Doi eintragen},
abstract = {Several molecular modifications accumulate in the human
organism with increasing age. Some of these „molecular
clocks“ in DNA and in proteins open up promising
approaches for the development of methods for forensic age
estimation. A natural limitation of these methods arises
from the fact that the chronological age is determined only
indirectly by analyzing defined molecular changes that occur
during aging. These changes are not linked exclusively to
the expired life span but may be influenced significantly by
intrinsic and extrinsic factors in the complex process of
individual aging. We tested the hypothesis that a combined
use of different “molecular clocks” in different tissues
results in more precise age estimates because this approach
addresses the complex ageing processes in a more
comprehensive way. Two molecular clocks (accumulation of
D-aspartic acid (D-Asp), accumulation of pentosidine (PEN))
in two different tissues (annulus fibrosus of intervertebral
discs and elastic cartilage of the epiglottis) were analyzed
in 95 cases, and uni- and multivariate models for age
estimation were generated.The more parameters were included
in the models for age estimation, the smaller the mean
absolute errors (MAE) became. While the MAEs were 7.5 –
11.0 years in univariate models, a multivariate model based
on the two protein clocks in the two tissues resulted in a
MAE of 4.0 years. These results support our hypothesis. The
tested approach of a combined analysis of different
molecular clocks analyzed in different tissues opens up new
possibilities in postmortem age estimation. In a next step,
we will add the epigenetic clock (DNA methylation) to our
protein clocks (PEN, D-Asp) and expand our set of
tissues.Keywords: Age estimation, pentosidine, D-aspartic
acid, machine learning, age prediction model, molecular
clocks},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {574 - Theory, modelling and simulation (POF3-574)},
pid = {G:(DE-HGF)POF3-574},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30976985},
UT = {WOS:000516579000041},
doi = {10.1007/s00414-019-02054-9},
url = {https://juser.fz-juelich.de/record/861914},
}