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@ARTICLE{Poeppl:861955,
author = {Poeppl, Timm B. and Langguth, Berthold and Laird, Angela R.
and Eickhoff, Simon},
title = {{M}eta-analytic {E}vidence for {N}eural {D}ysactivity
{U}nderlying {S}exual {D}ysfunction},
journal = {The journal of sexual medicine},
volume = {16},
number = {5},
issn = {1743-6095},
address = {[London]},
publisher = {Elsevier},
reportid = {FZJ-2019-02360},
pages = {614–617},
year = {2019},
note = {Funding: Angela R. Laird was supported by awards from the
National Institutes of Mental Health (U01 DA041156 and R01
DA041353) and the National Science Foundation (1631325 and
CNS 1532061). Simon B. Eickhoff was supported by the
Deutsche Forschungsgemeinschaft (DFG; EI 816/4–1, EI
816/6–1), the National Institute of Mental Health (R01
MH074457), the Helmholtz Portfolio Theme "Supercomputing and
Modeling for the Human Brain" and the European Union's
Horizon 2020 Research and Innovation Programme under Grant
Agreement No. 7202070 (HBP SGA1).},
abstract = {About $30-40\%$ of the population report sexual
dysfunction. Although it is well known that the brain
controls sexual behavior, little is known about the neural
basis of sexual dysfunction.AIM:To assess convergence of
altered brain activity associated with sexual dysfunction
across available functional imaging studies.METHODS:We used
activation likelihood estimation meta-analysis to quantify
interstudy concordance across 14 functional imaging studies
reporting 179 foci from 40 individual analyses involving 191
subjects with sexual dysfunction and 123 controls.MAIN
OUTCOME MEASURE:Activation likelihood estimation scores were
used to assess convergence of findings.RESULTS:Consistently
decreased brain activity associated with sexual dysfunction
was identified in the dorsal anterior cingulate cortex,
ventral striatum, dorsal midbrain, anterior midcingulate
cortex, and lateral orbitofrontal cortex.CLINICAL
IMPLICATION:These findings can serve as a basis for further
studies on the pathophysiology of this highly common
disorder with the view to development of more-specific
treatment strategies.STRENGTH $\&$ LIMITATIONS:Findings are
based on an observer-independent meta-analysis that provides
robust evidence for and anatomic localization of altered
brain activity related to sexual dysfunction. Our analysis
cannot distinguish between the putative sources of sexual
dysfunction, but it provides a more ubiquitous and general
pattern of related altered neural activity.CONCLUSION:The
identified regions have previously been shown to be
critically involved in mediating sexual arousal and to be
part of the sympathetic division of the autonomic nervous
system. This suggests that the disturbance of brain activity
associated with sexual dysfunction primarily affects sexual
arousal already at early stages that are controlled by the
sympathetic nervous system. Poeppl TB, Langguth B, Laird AR,
et al. Meta-analytic Evidence for Neural Dysactivity
Underlying Sexual Dysfunction. J Sex Med 2019;XX:XXX-XXX.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572) / SMHB -
Supercomputing and Modelling for the Human Brain
(HGF-SMHB-2013-2017) / HBP SGA1 - Human Brain Project
Specific Grant Agreement 1 (720270)},
pid = {G:(DE-HGF)POF3-572 / G:(DE-Juel1)HGF-SMHB-2013-2017 /
G:(EU-Grant)720270},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30926513},
UT = {WOS:000469936400002},
doi = {10.1016/j.jsxm.2019.02.012},
url = {https://juser.fz-juelich.de/record/861955},
}