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@ARTICLE{Sachs:862492,
author = {Sachs, Julia and Döhl, Katja and Weber, Anja and Bonus,
Michele and Ehlers, Ferdinand and Fleischer, Edmond and
Klinger, Anette and Gohlke, Holger and Pietruszka, Jörg and
Schmitt, Lutz and Teusch, Nicole},
title = {{N}ovel 3,4-{D}ihydroisocoumarins {I}nhibit {H}uman {P}-gp
and {BCRP} in {M}ultidrug {R}esistant {T}umors and
{D}emonstrate {S}ubstrate {I}nhibition of {Y}east {P}dr5},
journal = {Frontiers in pharmacology},
volume = {10},
issn = {1663-9812},
address = {Lausanne},
publisher = {Frontiers Media},
reportid = {FZJ-2019-02798},
pages = {400},
year = {2019},
abstract = {Multidrug resistance (MDR) in tumors and pathogens remains
a major problem in the efficacious treatment of patients by
reduction of therapy options and subsequent treatment
failure. Various mechanisms are described to be involved in
the development of MDR with overexpression of ATP-binding
cassette (ABC) transporters reflecting the most extensively
studied. These membrane transporters translocate a wide
variety of substrates utilizing energy from ATP hydrolysis
leading to decreased intracellular drug accumulation and
impaired drug efficacy. One treatment strategy might be
inhibition of transporter-mediated efflux by small
molecules. Isocoumarins and 3,4-dihydroisocoumarins are a
large group of natural products derived from various sources
with great structural and functional variety, but have so
far not been in the focus as potential MDR reversing agents.
Thus, three natural products and nine novel
3,4-dihydroisocoumarins were designed and analyzed regarding
cytotoxicity induction and inhibition of human ABC
transporters P-glycoprotein (P-gp), multidrug
resistance-associated protein 1 (MRP1) and breast cancer
resistance protein (BCRP) in a variety of human cancer cell
lines as well as the yeast ABC transporter Pdr5 in
Saccharomyces cerevisiae. Dual inhibitors of P-gp and BCRP
and inhibitors of Pdr5 were identified, and distinct
structure-activity relationships for transporter inhibition
were revealed. The strongest inhibitor of P-gp and BCRP,
which inhibited the transporters up to 80 to $90\%$ compared
to the respective positive controls, demonstrated the
ability to reverse chemotherapy resistance in resistant
cancer cell lines up to 5.6-fold. In the case of Pdr5,
inhibitors were identified that prevented substrate
transport and/or ATPase activity with IC50 values in the low
micromolar range. However, cell toxicity was not observed.
Molecular docking of the test compounds to P-gp revealed
that differences in inhibition capacity were based on
different binding affinities to the transporter. Thus, these
small molecules provide novel lead structures for further
optimization.},
cin = {JSC / ICS-6 / NIC / IBOC / IBG-1},
ddc = {610},
cid = {I:(DE-Juel1)JSC-20090406 / I:(DE-Juel1)ICS-6-20110106 /
I:(DE-Juel1)NIC-20090406 / I:(DE-Juel1)IBOC-20090406 /
I:(DE-Juel1)IBG-1-20101118},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511) / Forschergruppe Gohlke $(hkf7_20170501)$ / 581 -
Biotechnology (POF3-581)},
pid = {G:(DE-HGF)POF3-511 / $G:(DE-Juel1)hkf7_20170501$ /
G:(DE-HGF)POF3-581},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31040786},
UT = {WOS:000464599600001},
doi = {10.3389/fphar.2019.00400},
url = {https://juser.fz-juelich.de/record/862492},
}