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@ARTICLE{Dafsari:864151,
      author       = {Dafsari, Hormos Salimi and Dovern, Anna and Fink, Gereon R.
                      and Weiss-Blankenhorn, Peter},
      title        = {{D}eficient body structural description contributes to
                      apraxic end-position errors in imitation},
      journal      = {Neuropsychologia},
      volume       = {133},
      issn         = {0028-3932},
      address      = {Amsterdam [u.a.]},
      publisher    = {Elsevier Science},
      reportid     = {FZJ-2019-04021},
      pages        = {107150},
      year         = {2019},
      abstract     = {Apraxia is a common cognitive deficit after left hemisphere
                      (LH) stroke. It has been suggested that a disturbed
                      representation of the human body underlies apraxic imitation
                      deficits. Thus, we here tested the hypothesis that a
                      deficient body structural description (BSD), i.e., a
                      deficient representation of a body part's position (relative
                      to a standard human body), contributes to apraxic
                      end-position errors in imitation, while controlling for
                      deficits in the semantic representation of the human body
                      (body image, BI) and naming deficits.A quantitative pointing
                      task to assess putative BSD deficits and an apraxia
                      assessment, including imitation and pantomime tasks, were
                      applied to 27 patients with LH stroke and 19 healthy
                      subjects. While LH stroke patients without apraxia (n=15)
                      did not differ from control subjects in their pointing
                      performance, patients suffering from imitation apraxia
                      (n=10) showed a differential deficit when pointing to body
                      parts of other humans compared to object parts. Voxel-based
                      lesion symptom mapping (VLSM) revealed an association of
                      these differential pointing deficits (indicating a deficient
                      BSD) with lesions in the angular gyrus of the left inferior
                      parietal cortex.This first quantitative group study of BSD
                      deficits in LH stroke patients supports the notion that
                      apraxic end-position errors in imitation are – at least in
                      part – due to a deficient coding of the position of human
                      body parts.},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {572 - (Dys-)function and Plasticity (POF3-572)},
      pid          = {G:(DE-HGF)POF3-572},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:31369744},
      UT           = {WOS:000493915600002},
      doi          = {10.1016/j.neuropsychologia.2019.107150},
      url          = {https://juser.fz-juelich.de/record/864151},
}