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@ARTICLE{Krishnan:864627,
author = {Krishnan, Jeyashree and Torabi, Reza and Di Napoli, Edoardo
and Schuppert, Andreas},
title = {{A} {M}odified {I}sing {M}odel of {B}arabási-{A}lbert
{N}etwork with {G}ene-type {S}pins},
journal = {Journal of mathematical biology},
volume = {81},
issn = {0303-6812},
address = {New York},
publisher = {Springer},
reportid = {FZJ-2019-04332},
pages = {769–798},
year = {2020},
abstract = {The central question of systems biology is to understand
how individual components of a biological system such as
genes or proteins cooperate in emerging phenotypes resulting
in the evolution of diseases. As living cells are open
systems in quasi-steady state type equilibrium in continuous
exchange with their environment, computational techniques
that have been successfully applied in statistical
thermodynamics to describe phase transitions may provide new
insights to emerging behavior of biological systems. Here we
will systematically evaluate the translation of
computational techniques from solid-state physics to network
models that closely resemble biological networks and develop
specific translational rules to tackle problems unique to
living systems. Hence we will focus on logic models
exhibiting only two states in each network node. Motivated
by the apparent asymmetry between biological states where an
entity exhibits boolean states i.e. is active or inactive,
we present an adaptation of symmetric Ising model towards an
asymmetric one fitting to living systems here referred to as
the modified Ising model with gene-type spins. We analyze
phase transitions by Monte Carlo simulations and propose
mean-field solution of modified Ising model of a network
type that closely resembles real-world network, the
$Barab\'{a}si-Albert$ model of scale-free networks. We show
that asymmetric Ising models show similarities to symmetric
Ising models with external field and undergoes a
discontinuous phase transition of the first-order and
exhibits hysteresis. The simulation setup presented here can
be directly used for any biological network connectivity
dataset and is also applicable for other networks that
exhibit similar states of activity. This is a general
statistical method to deal with non-linear large scale
models arising in the context of biological systems and is
scalable to any network size.},
cin = {JSC},
ddc = {570},
cid = {I:(DE-Juel1)JSC-20090406},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511) / Simulation and Data Laboratory Quantum
Materials (SDLQM) (SDLQM)},
pid = {G:(DE-HGF)POF3-511 / G:(DE-Juel1)SDLQM},
typ = {PUB:(DE-HGF)16},
eprint = {1908.06872},
howpublished = {arXiv:1908.06872},
archivePrefix = {arXiv},
SLACcitation = {$\%\%CITATION$ = $arXiv:1908.06872;\%\%$},
pubmed = {pmid:32897406},
UT = {WOS:000567448200001},
doi = {10.1007/s00285-020-01518-6},
url = {https://juser.fz-juelich.de/record/864627},
}