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@ARTICLE{Hillringhaus:865247,
author = {Hillringhaus, Sebastian and Dasanna, Anil K. and Gompper,
Gerhard and Fedosov, Dmitry A.},
title = {{I}mportance of {E}rythrocyte {D}eformability for the
{A}lignment of {M}alaria {P}arasite upon {I}nvasion},
journal = {Biophysical journal},
volume = {117},
number = {7},
issn = {0006-3495},
address = {Bethesda, Md.},
publisher = {Soc.},
reportid = {FZJ-2019-04771},
pages = {1202-1214},
year = {2019},
abstract = {Invasion of erythrocytes by merozoites is an essential step
for the survival and progression of malaria parasites. To
invade red blood cells (RBCs), apicomplexan parasites have
to adhere with their apex to the RBC membrane. This
necessary apex-membrane contact (or alignment) is not
immediately established because the orientation of a free
merozoite with respect to the RBC membrane is random when an
adhesion contact first occurs. Therefore, it has been
suggested that after the initial adhesion, merozoites
facilitate their proper alignment by inducing considerable
membrane deformations, frequently observed before the
invasion process. This proposition is based on a positive
correlation between RBC membrane deformation and successful
invasion; however, the role of RBC mechanics and its
deformation in the alignment process remains elusive. Using
a mechanically realistic model of a deformable RBC, we
investigate numerically the importance of RBC deformability
for merozoite alignment. Adhesion between the parasite and
RBC membrane is modeled by an attractive potential that
might be inhomogeneous, mimicking possible adhesion
gradients at the surface of a parasite. Our results show
that RBC membrane deformations are crucial for successful
merozoite alignment and require interaction strengths
comparable to adhesion forces measured experimentally.
Adhesion gradients along the parasite body further improve
its alignment. Finally, an increased membrane rigidity is
found to result in poor merozoite alignment, which can be a
possible reason for a reduction in the invasion
susceptibility of RBCs in several blood diseases associated
with membrane stiffening.},
cin = {ICS-2 / JARA-HPC},
ddc = {570},
cid = {I:(DE-Juel1)ICS-2-20110106 / $I:(DE-82)080012_20140620$},
pnm = {552 - Engineering Cell Function (POF3-552) / Formation of
Polymer-Particle Aggregates in Blood Flow
$(jiff44_20180501)$},
pid = {G:(DE-HGF)POF3-552 / $G:(DE-Juel1)jiff44_20180501$},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31540708},
UT = {WOS:000488457600004},
doi = {10.1016/j.bpj.2019.08.027},
url = {https://juser.fz-juelich.de/record/865247},
}