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@ARTICLE{Hilgers:865990,
author = {Hilgers, Fabienne and Bitzenhofer, Nora Lisa and Ackermann,
Yannic and Burmeister, Alina and Grünberger, Alexander and
Jaeger, Karl-Erich and Drepper, Thomas},
title = {{G}enetically encoded photosensitizers as light-triggered
antimicrobial agents},
journal = {International journal of molecular sciences},
volume = {20},
number = {18},
issn = {1422-0067},
address = {Basel},
publisher = {Molecular Diversity Preservation International},
reportid = {FZJ-2019-05251},
pages = {4608 -},
year = {2019},
abstract = {Diseases caused by multi-drug resistant pathogens have
become a global concern. Therefore, new approaches suitable
for treating these bacteria are urgently needed. In this
study, we analyzed genetically encoded photosensitizers (PS)
related to the green fluorescent protein (GFP) or
light-oxygen-voltage (LOV) photoreceptors for their
exogenous applicability as light-triggered antimicrobial
agents. Depending on their specific photophysical properties
and photochemistry, these PSs can produce different toxic
ROS (reactive oxygen species) such as O2•− and H2O2 via
type-I, as well as 1O2 via type-II reaction in response to
light. By using cell viability assays and microfluidics, we
could demonstrate differences in the intracellular and
extracellular phototoxicity of the applied PS. While
intracellular expression and exogenous supply of GFP-related
PSs resulted in a slow inactivation of E. coli and
pathogenic Gram-negative and Gram-positive bacteria,
illumination of LOV-based PSs such as the singlet oxygen
photosensitizing protein SOPP3 resulted in a fast and
homogeneous killing of these microbes. Furthermore, our data
indicate that the ROS type and yield as well as the
localization of the applied PS protein can strongly
influence the antibacterial spectrum and efficacy. These
findings open up new opportunities for photodynamic
inactivation of pathogenic bacteria},
cin = {IMET / IBG-1},
ddc = {540},
cid = {I:(DE-Juel1)IMET-20090612 / I:(DE-Juel1)IBG-1-20101118},
pnm = {581 - Biotechnology (POF3-581)},
pid = {G:(DE-HGF)POF3-581},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31533368},
UT = {WOS:000489100500287},
doi = {10.3390/ijms20184608},
url = {https://juser.fz-juelich.de/record/865990},
}