000872767 001__ 872767 000872767 005__ 20210130004303.0 000872767 0247_ $$2doi$$a10.1039/C8CS00034D 000872767 0247_ $$2ISSN$$a0306-0012 000872767 0247_ $$2ISSN$$a1460-4744 000872767 0247_ $$2altmetric$$aaltmetric:62509571 000872767 0247_ $$2pmid$$apmid:31192324 000872767 0247_ $$2WOS$$aWOS:000475647600006 000872767 037__ $$aFZJ-2020-00245 000872767 082__ $$a540 000872767 1001_ $$00000-0001-9076-7114$$aOwen, Michael C.$$b0 000872767 245__ $$aEffects of in vivo conditions on amyloid aggregation 000872767 260__ $$aLondon$$bSoc.$$c2019 000872767 3367_ $$2DRIVER$$aarticle 000872767 3367_ $$2DataCite$$aOutput Types/Journal article 000872767 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1580896971_10825 000872767 3367_ $$2BibTeX$$aARTICLE 000872767 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000872767 3367_ $$00$$2EndNote$$aJournal Article 000872767 520__ $$aOne of the grand challenges of biophysical chemistry is to understand the principles that govern protein misfolding and aggregation, which is a highly complex process that is sensitive to initial conditions, operates on a huge range of length- and timescales, and has products that range from protein dimers to macroscopic amyloid fibrils. Aberrant aggregation is associated with more than 25 diseases, which include Alzheimer's, Parkinson's, Huntington's, and type II diabetes. Amyloid aggregation has been extensively studied in the test tube, therefore under conditions that are far from physiological relevance. Hence, there is dire need to extend these investigations to in vivo conditions where amyloid formation is affected by a myriad of biochemical interactions. As a hallmark of neurodegenerative diseases, these interactions need to be understood in detail to develop novel therapeutic interventions, as millions of people globally suffer from neurodegenerative disorders and type II diabetes. The aim of this review is to document the progress in the research on amyloid formation from a physicochemical perspective with a special focus on the physiological factors influencing the aggregation of the amyloid-β peptide, the islet amyloid polypeptide, α-synuclein, and the hungingtin protein. 000872767 536__ $$0G:(DE-HGF)POF3-553$$a553 - Physical Basis of Diseases (POF3-553)$$cPOF3-553$$fPOF III$$x0 000872767 588__ $$aDataset connected to CrossRef 000872767 7001_ $$00000-0001-9393-503X$$aGnutt, David$$b1 000872767 7001_ $$0P:(DE-HGF)0$$aGao, Mimi$$b2 000872767 7001_ $$0P:(DE-HGF)0$$aWärmländer, Sebastian K. T. 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