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@ARTICLE{Strodel:872769,
author = {Strodel, Birgit and Coskuner-Weber, Orkid},
title = {{T}ransition {M}etal {I}on {I}nteractions with {D}isordered
{A}myloid-β {P}eptides in the {P}athogenesis of
{A}lzheimer’s {D}isease: {I}nsights from {C}omputational
{C}hemistry {S}tudies},
journal = {Journal of chemical information and modeling},
volume = {59},
number = {5},
issn = {1549-960X},
address = {Washington, DC},
publisher = {American Chemical Society64160},
reportid = {FZJ-2020-00247},
pages = {1782 - 1805},
year = {2019},
abstract = {Monomers and oligomers of the amyloid-β peptide aggregate
to form the fibrils found in the brains of Alzheimer’s
disease patients. These monomers and oligomers are largely
disordered and can interact with transition metal ions,
affecting the mechanism and kinetics of amyloid-β
aggregation. Due to the disordered nature of amyloid-β, its
rapid aggregation, as well as solvent and paramagnetic
effects, experimental studies face challenges in the
characterization of transition metal ions bound to
amyloid-β monomers and oligomers. The details of the
coordination chemistry between transition metals and
amyloid-β obtained from experiments remain debated.
Furthermore, the impact of transition metal ion binding on
the monomeric or oligomeric amyloid-β structures and
dynamics are still poorly understood. Computational
chemistry studies can serve as an important complement to
experimental studies and can provide additional knowledge on
the binding between amyloid-β and transition metal ions.
Many research groups conducted first-principles
calculations, ab initio molecular dynamics simulations,
quantum mechanics/classical mechanics simulations, and
classical molecular dynamics simulations for studying the
interplay between transition metal ions and amyloid-β
monomers and oligomers. This review summarizes the current
understanding of transition metal interactions with
amyloid-β obtained from computational chemistry studies. We
also emphasize the current view of the coordination
chemistry between transition metal ions and amyloid-β. This
information represents an important foundation for future
metal ion chelator and drug design studies aiming to combat
Alzheimer’s disease.},
cin = {ICS-6},
ddc = {540},
cid = {I:(DE-Juel1)ICS-6-20110106},
pnm = {553 - Physical Basis of Diseases (POF3-553)},
pid = {G:(DE-HGF)POF3-553},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30933519},
UT = {WOS:000469884900011},
doi = {10.1021/acs.jcim.8b00983},
url = {https://juser.fz-juelich.de/record/872769},
}