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@ARTICLE{Viegas:874256,
author = {Viegas, Aldino and Dollinger, Peter and Verma, Neha and
Kubiak, Jakub and Viennet, Thibault and Seidel, Claus A. M.
and Gohlke, Holger and Etzkorn, Manuel and Kovacic, Filip
and Jaeger, Karl-Erich},
title = {{S}tructural and dynamic insights revealing how lipase
binding domain {MD}1 of {P}seudomonas aeruginosa foldase
affects lipase activation},
journal = {Scientific reports},
volume = {10},
number = {1},
issn = {2045-2322},
address = {[London]},
publisher = {Macmillan Publishers Limited, part of Springer Nature},
reportid = {FZJ-2020-01347},
pages = {3578},
year = {2020},
abstract = {Folding and cellular localization of many proteins of
Gram-negative bacteria rely on a network of chaperones and
secretion systems. Among them is the lipase-specific foldase
Lif, a membrane-bound steric chaperone that tightly binds
(KD = 29 nM) and mediates folding of the lipase LipA,
a virulence factor of the pathogenic bacterium P.
aeruginosa. Lif consists of five-domains, including a mini
domain MD1 essential for LipA folding. However, the
molecular mechanism of Lif-assisted LipA folding remains
elusive. Here, we show in in vitro experiments using a
soluble form of Lif (sLif) that isolated MD1 inhibits
sLif-assisted LipA activation. Furthermore, the ability to
activate LipA is lost in the variant sLifY99A, in which the
evolutionary conserved amino acid Y99 from helix α1 of MD1
is mutated to alanine. This coincides with an approximately
three-fold reduced affinity of the variant to LipA together
with increased flexibility of sLifY99A in the complex as
determined by polarization-resolved fluorescence
spectroscopy. We have solved the NMR solution structures of
P. aeruginosa MD1 and variant MD1Y99A revealing a similar
fold indicating that a structural modification is likely not
the reason for the impaired activity of variant sLifY99A.
Molecular dynamics simulations of the sLif:LipA complex in
connection with rigidity analyses suggest a long-range
network of interactions spanning from Y99 of sLif to the
active site of LipA, which might be essential for LipA
activation. These findings provide important details about
the putative mechanism for LipA activation and point to a
general mechanism of protein folding by multi-domain steric
chaperones.},
cin = {JSC / NIC / ICS-6 / IBG-1 / IMET},
ddc = {600},
cid = {I:(DE-Juel1)JSC-20090406 / I:(DE-Juel1)NIC-20090406 /
I:(DE-Juel1)ICS-6-20110106 / I:(DE-Juel1)IBG-1-20101118 /
I:(DE-Juel1)IMET-20090612},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511) / Forschergruppe Gohlke $(hkf7_20170501)$ /
Conformational dynamics of the unbound lipase-specific
foldase Lif $(hdd16_20161101)$ / Analysis of the
conformational changes during activation of lipase A by its
foldase $(hdd16_20171101)$ / 581 - Biotechnology (POF3-581)},
pid = {G:(DE-HGF)POF3-511 / $G:(DE-Juel1)hkf7_20170501$ /
$G:(DE-Juel1)hdd16_20161101$ / $G:(DE-Juel1)hdd16_20171101$
/ G:(DE-HGF)POF3-581},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32107397},
UT = {WOS:000563068600012},
doi = {10.1038/s41598-020-60093-4},
url = {https://juser.fz-juelich.de/record/874256},
}