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@ARTICLE{Yu:874556,
author = {Yu, Qingfen and Dasgupta, Sabyasachi and Auth, Thorsten and
Gompper, Gerhard},
title = {{O}smotic {C}oncentration-{C}ontrolled {P}article {U}ptake
and {W}rapping-{I}nduced {L}ysis of {C}ells and {V}esicles},
journal = {Nano letters},
volume = {20},
number = {3},
issn = {1530-6992},
address = {Washington, DC},
publisher = {ACS Publ.},
reportid = {FZJ-2020-01510},
pages = {1662 - 1668},
year = {2020},
abstract = {In vivo, high protein and ion concentrations determine the
preferred volumes of cells, organelles, and vesicles.
Deformations of their lipid-bilayer membranes by
nanoparticle wrapping reduce the interior volumes available
to solutes and thus induce large osmotic pressure
differences. Osmotic concentration can therefore be an
important control parameter for wrapping of nanoparticles.
We employ a curvature-elasticity model of the membrane and
contact interaction with spherical particles to study their
wrapping at initially spherical vesicles. Although the
continuous particle-binding transition is independent of the
presence of solutes, the discontinuous envelopment
transition shifts to higher adhesion strengths and the
corresponding energy barrier increases with increasing
osmotic concentration. High osmotic concentrations stabilize
partial-wrapped, membrane-bound states for both, particle
attachment to the inside and the outside. In this regime,
wrapping of particles controls membrane tension, with
power-law dependencies on osmotic concentration and adhesion
strength. For high adhesion strengths, particle wrapping can
lead to the opening of mechanosensitive channels in cell
membranes and to lysis. Membrane tension-induced
stabilization of partial-wrapped states as well as
wrapping-induced lysis play important roles not only for
desired mechano-bacteriocidal effects of engineered
nanomaterials but may also determine viral burst sizes of
bacteria and control endocytosis for mammalian cells.},
cin = {IBI-5},
ddc = {660},
cid = {I:(DE-Juel1)IBI-5-20200312},
pnm = {552 - Engineering Cell Function (POF3-552)},
pid = {G:(DE-HGF)POF3-552},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32046489},
UT = {WOS:000526408800025},
doi = {10.1021/acs.nanolett.9b04788},
url = {https://juser.fz-juelich.de/record/874556},
}