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@ARTICLE{Sripad:874650,
      author       = {Sripad, Praveen and Rosenberg, Jessica and Boers, Frank and
                      Filss, Christian P. and Galldiks, Norbert and Langen,
                      Karl-Josef and Clauss, Ralf and Shah, N. Jon and Dammers,
                      Jürgen},
      title        = {{E}ffect of {Z}olpidem in the {A}ftermath of {T}raumatic
                      {B}rain {I}njury: {A}n {MEG} {S}tudy},
      journal      = {Case reports in neurological medicine},
      volume       = {2020},
      issn         = {2090-6676},
      address      = {New York, NY},
      publisher    = {Hindawi},
      reportid     = {FZJ-2020-01568},
      pages        = {1 - 8},
      year         = {2020},
      abstract     = {In the past two decades, many studies have shown the
                      paradoxical efficacy of zolpidem, a hypnotic used to induce
                      sleep, in transiently alleviating various disorders of
                      consciousness such as traumatic brain injury (TBI),
                      dystonia, and Parkinson’s disease. The mechanism of action
                      of this effect of zolpidem is of great research interest. In
                      this case study, we use magnetoencephalography (MEG) to
                      investigate a fully conscious, ex-coma patient who suffered
                      from neurological difficulties for a few years due to
                      traumatic brain injury. For a few years after injury, the
                      patient was under medication with zolpidem that drastically
                      improved his symptoms. MEG recordings taken before and after
                      zolpidem showed a reduction in power in the theta-alpha
                      (4–12 Hz) and lower beta (15–20 Hz) frequency bands.
                      An increase in power after zolpidem intake was found in the
                      higher beta/lower gamma (20–43 Hz) frequency band.
                      Source level functional connectivity measured using
                      weighted-phase lag index showed changes after zolpidem
                      intake. Stronger connectivity between left frontal and
                      temporal brain regions was observed. We report that zolpidem
                      induces a change in MEG resting power and functional
                      connectivity in the patient. MEG is an informative and
                      sensitive tool to detect changes in brain activity for TBI.},
      cin          = {INM-4 / INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-4-20090406 / I:(DE-Juel1)INM-3-20090406},
      pnm          = {572 - (Dys-)function and Plasticity (POF3-572)},
      pid          = {G:(DE-HGF)POF3-572},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:32257474},
      UT           = {WOS:000522949400001},
      doi          = {10.1155/2020/8597062},
      url          = {https://juser.fz-juelich.de/record/874650},
}