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@ARTICLE{Giehl:889195,
author = {Giehl, Kathrin and Ophey, Anja and Hammes, Jochen and
Rehberg, Sarah and Lichtenstein, Thorsten and Reker, Paul
and Eggers, Carsten and Kalbe, Elke and van Eimeren, Thilo},
title = {{W}orking memory training increases neural efficiency in
{P}arkinson’s disease: a randomized controlled trial},
journal = {Brain communications},
volume = {2},
number = {2},
issn = {2632-1297},
address = {[GroÃbritannien]},
publisher = {Guarantors of Brain},
reportid = {FZJ-2021-00105},
pages = {fcaa115},
year = {2020},
abstract = {Impairment of working memory and executive functions is
already frequently observed in early stages of Parkinson’s
disease. Improvements in working memory performance in this
cohort could potentially be achieved via working memory
training. However, the specific neural mechanisms underlying
different working memory processes such as maintenance as
opposed to manipulation are largely under-investigated in
Parkinson’s disease. Moreover, the plasticity of these
correlates as a function of working memory training is
currently unknown in this population. Thus, the working
memory subprocesses of maintenance and manipulation were
assessed in 41 cognitively healthy patients with
Parkinson’s disease using a newly developed working memory
paradigm and functional MRI. Nineteen patients were
randomized to a 5-week home-based digital working memory
training intervention while the remaining patients entered a
control, wait list condition. Working memory task-related
activation patterns and context-dependent functional
connectivity, as well as the change of these neural
correlates as a function of training, were assessed. While
both working memory processes activated an extended
frontoparietal–cerebellar network, only the manipulation
of items within working memory also recruited the anterior
striatum. The intervention effect on the neural correlates
was small, but decreased activation in areas relevant for
working memory could be observed, with activation changes
correlating with behavioural change. Moreover, training
seemed to result in decreased functional connectivity when
pure maintenance was required, and in a reorganization of
functional connectivity when items had to be manipulated. In
accordance with the neural efficacy hypothesis, training
resulted in overall reduced activation and reorganized
functional connectivity, with a differential effect on the
different working memory processes under investigation. Now,
larger trials including follow-up examinations are needed to
further explore the long-term effects of such interventions
on a neural level and to estimate the clinical relevance to
potentially delay cognitive decline in cognitively healthy
patients with Parkinson’s disease.Keywords: idiopathic
Parkinson’s disease, home-based working memory training,
functional magnetic resonance imaging, blood oxygen level
dependent signal, functional connectivity},
cin = {INM-2},
ddc = {610},
cid = {I:(DE-Juel1)INM-2-20090406},
pnm = {89573 - Neuroimaging (POF2-89573)},
pid = {G:(DE-HGF)POF2-89573},
typ = {PUB:(DE-HGF)16},
pubmed = {32954349},
UT = {WOS:000639431800056},
doi = {10.1093/braincomms/fcaa115},
url = {https://juser.fz-juelich.de/record/889195},
}