% IMPORTANT: The following is UTF-8 encoded.  This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.

@ARTICLE{Stickel:889303,
      author       = {Stickel, S. and Eickhoff, Simon and Habel, U. and
                      Stickeler, E. and Goecke, T. W. and Lang, J. and Chechko,
                      N.},
      title        = {{E}ndocrine stress response in pregnancy and 12 weeks
                      postpartum – {E}xploring risk factors for postpartum
                      depression},
      journal      = {Psychoneuroendocrinology},
      volume       = {125},
      issn         = {0306-4530},
      address      = {Amsterdam [u.a.]},
      publisher    = {Elsevier Science},
      reportid     = {FZJ-2021-00195},
      pages        = {105122},
      year         = {2021},
      note         = {This study was funded by the rotation program (2015–2017)
                      of the medical faculty of the University Hospital RWTH
                      Aachen, the Deutsche Forschungsgemeinschaft (German Research
                      Foundation, DFG, CH 1718/2-1) and the International Research
                      Training Group (IRTG 2150) of the DFG.},
      abstract     = {Pregnancy and the postpartum period are characterized by
                      physiological alterations in cortisol and cortisone levels.
                      In the present study, we sought to explore the risk factors
                      for postpartum depression (PPD) and self-remitting
                      postpartum adjustment disorder (AD) and whether
                      cortisol/cortisone metabolism might have any bearing on
                      them. Hair samples from 196 participants (mean age = 31.44,
                      SD = 4.71) were collected at two time points (1-6 days after
                      childbirth and 12 weeks postpartum) to determine the
                      cumulative hair cortisol (HCC) and hair cortisone (HCNC)
                      exposure in the third trimester and during the 12 weeks
                      postpartum. Compared to the non-depressed group (ND, n =
                      141), more women in the AD (n = 28) and PPD (n = 27) groups
                      had a personal or family history of depression and more
                      stressful life events. Compared to ND and PPD, more women in
                      the AD group had birth-related complications with their
                      children being more often transferred to a pediatric ward.
                      The factors associated with PPD were found to include being
                      unmarried and having a lower household income, less support
                      at home, more subjectively perceived stress after childbirth
                      and lower maternal sensitivity. The natural decrease in HCC
                      concentration from the third trimester to 12 weeks
                      postpartum was significant only in the ND and AD groups, but
                      not in PPD. In summary, prolonged subjectively perceived
                      postpartum stress associated with living situations may
                      contribute to the development of PPD while birth- and
                      child-related complications are likely to trigger brief
                      episodes of AD. Only in ND and AD, the pregnancy-related
                      physiological changes in glucocorticoid levels return to the
                      pre-pregnancy baseline after 12 weeks. Our observations
                      point to the difference between the ND and PPD groups in
                      glucocorticoid metabolism-related postpartum adjustment,
                      which may be a factor in the development of PPD.},
      cin          = {INM-7 / INM-10},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-7-20090406 / I:(DE-Juel1)INM-10-20170113},
      pnm          = {525 - Decoding Brain Organization and Dysfunction
                      (POF4-525)},
      pid          = {G:(DE-HGF)POF4-525},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {33421704},
      UT           = {WOS:000621359000012},
      doi          = {10.1016/j.psyneuen.2020.105122},
      url          = {https://juser.fz-juelich.de/record/889303},
}