% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Schnakenberg:890668,
author = {Schnakenberg, Patricia and Jo, Han-Gue and Stickel, Susanne
and Habel, Ute and Eickhoff, Simon B. and Brodkin, Edward S.
and Goecke, Tamme Weyert and Votinov, Mikhail and Chechko,
Natalia},
title = {{T}he early postpartum period – {D}ifferences between
women with and without a history of depression},
journal = {Anatomical science international},
volume = {136},
issn = {0022-3956},
address = {Tokyo},
publisher = {Springer814959},
reportid = {FZJ-2021-01116},
pages = {109-116},
year = {2021},
abstract = {Depression is a highly recurrent disorder. When in
remission, it affords an important opportunity to understand
the state-independent neurobiological alterations, as well
as the socio-demographic characteristics, that likely
contribute to the recurrence of major depressive disorder
(MDD). The present study examined 110 euthymic women in
their early postpartum period. A comparison was made between
participants with (n = 20) and without (n = 90) a history of
MDD by means of a multimodal approach including an fMRI
experiment, assessment of hair cortisol concentration (HCC)
and a clinical anamnestic interview. Women with a personal
history of MDD were found to have decreased resting-state
functional connectivity (RSFC) between the lateral parietal
cortex (LPC) and the posterior cingulate cortex (PCC), and
their Edinburgh Postnatal Depression Scale (EPDS) scores
were significantly higher shortly after childbirth. More
often than not, these women also had a family history of
MDD. While women with no history of depression showed a
negative association between hair cortisol concentration
(HCC) and gray matter volume (GMV) in the medial
orbitofrontal cortex (mOFC), the opposite trend was seen in
women with a history of depression. This implies that women
with remitted depression show distinctive neural phenotypes
with subclinical residual symptoms, which likely predispose
them to later depressive episodes.},
cin = {INM-7 / INM-10},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406 / I:(DE-Juel1)INM-10-20170113},
pnm = {525 - Decoding Brain Organization and Dysfunction
(POF4-525)},
pid = {G:(DE-HGF)POF4-525},
typ = {PUB:(DE-HGF)16},
pubmed = {33588224},
UT = {WOS:000634104500015},
doi = {10.1016/j.jpsychires.2021.01.056},
url = {https://juser.fz-juelich.de/record/890668},
}