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@ARTICLE{Schneider:891082,
author = {Schneider, Daniela and Bier, Dirk and Holschbach, Marcus
and Bauer, Andreas and Neumaier, Bernd},
title = {{S}pecies {D}ifferences in {M}icrosomal {M}etabolism of
{X}anthine-{D}erived {A}1 {A}denosine {R}eceptor {L}igands},
journal = {Pharmaceuticals},
volume = {14},
number = {3},
issn = {1424-8247},
address = {Basel},
publisher = {MDPI},
reportid = {FZJ-2021-01353},
pages = {277 -},
year = {2021},
abstract = {Tracer development for positron emission tomography (PET)
requires thorough evaluation of pharmacokinetics,
metabolism, and dosimetry of candidate radioligands in
preclinical animal studies. Since variations in
pharmacokinetics and metabolism of a compound occur in
different species, careful selection of a suitable model
species is mandatory to obtain valid data. This study
focuses on species differences in the in vitro metabolism of
three xanthine-derived ligands for the A1 adenosine receptor
(A1AR), which, in their 18F-labeled form, can be used to
image A1AR via PET. In vitro intrinsic clearance and
metabolite profiles of
8-cyclopentyl-3-(3-fluoropropyl)-1-propylxanthine (CPFPX),
an established A1AR-ligand, and two novel analogs,
8-cyclobutyl-3-(3-fluoropropyl)-1-propylxanthine (CBX) and
3-(3-fluoropropyl)-8-(1-methylcyclobutyl)-1-propylxanthine
(MCBX), were determined in liver microsomes from humans and
preclinical animal species. Molecular mechanisms leading to
significant differences between human and animal metabolite
profiles were also examined. The results revealed
significant species differences regarding qualitative and
quantitative aspects of microsomal metabolism. None of the
tested animal species fully matched human microsomal
metabolism of the three A1AR ligands. In conclusion,
preclinical evaluation of xanthine-derived A1AR ligands
should employ at least two animal species, preferably rodent
and dog, to predict in vivo behavior in humans.
Surprisingly, rhesus macaques appear unsuitable due to large
differences in metabolic activity towards the test
compounds.},
cin = {INM-5 / INM-2},
ddc = {610},
cid = {I:(DE-Juel1)INM-5-20090406 / I:(DE-Juel1)INM-2-20090406},
pnm = {525 - Decoding Brain Organization and Dysfunction
(POF4-525)},
pid = {G:(DE-HGF)POF4-525},
typ = {PUB:(DE-HGF)16},
pubmed = {33803861},
UT = {WOS:000634083300001},
doi = {10.3390/ph14030277},
url = {https://juser.fz-juelich.de/record/891082},
}