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@ARTICLE{Vanasse:891104,
author = {Vanasse, Thomas J. and Fox, Peter T. and Fox, P. Mickle and
Cauda, Franco and Costa, Tommaso and Smith, Stephen M. and
Eickhoff, Simon B. and Lancaster, Jack L.},
title = {{B}rain pathology recapitulates physiology: {A} network
meta-analysis},
journal = {Communications biology},
volume = {4},
number = {1},
issn = {2399-3642},
address = {London},
publisher = {Springer Nature},
reportid = {FZJ-2021-01367},
pages = {301},
year = {2021},
note = {This work was supported by the National Institute of Mental
Health of the U.S. NationalInstitute of Health under award
number R01 MH074457; the National Institute ofNeurological
Disorders and Stroke of the U.S. National Institutes of
Health under AwardNumber F32 NS114034; the U.S. Department
of Defense, Defense Health Program,Psychological Health and
Traumatic Brain Injury Research Program under the
Consortiumto Alleviate PTSD (CAP) award number
W81XWH-13-2-0065; and the U.S.Department of Veterans
Affairs, Office of Research $\&$ Development, Clinical
ScienceResearch $\&$ Development Service under award numer
I01CX001136-01. The content issolely the responsibility of
the authors and does not necessarily represent the
officialviews of these funding agencies.},
abstract = {Network architecture is a brain-organizational motif
present across spatial scales from cell assemblies to
distributed systems. Structural pathology in some
neurodegenerative disorders selectively afflicts a subset of
functional networks, motivating the network degeneration
hypothesis (NDH). Recent evidence suggests that structural
pathology recapitulating physiology may be a general
property of neuropsychiatric disorders. To test this
possibility, we compared functional and structural network
meta-analyses drawing upon the BrainMap database. The
functional meta-analysis included results from >7,000
experiments of subjects performing >100 task paradigms; the
structural meta-analysis included >2,000 experiments of
patients with >40 brain disorders. Structure-function
network concordance was high: $68\%$ of networks matched
(pFWE < 0.01), confirming the broader scope of NDH. This
correspondence persisted across higher model orders. A
positive linear association between disease and behavioral
entropy (p = 0.0006;R2 = 0.53) suggests nodal stress as a
common mechanism. Corroborating this interpretation with
independent data, we show that metabolic 'cost'
significantly differs along this transdiagnostic/multimodal
gradient.},
cin = {INM-7},
ddc = {570},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {525 - Decoding Brain Organization and Dysfunction
(POF4-525)},
pid = {G:(DE-HGF)POF4-525},
typ = {PUB:(DE-HGF)16},
pubmed = {33686216},
UT = {WOS:000627440400002},
doi = {10.1038/s42003-021-01832-9},
url = {https://juser.fz-juelich.de/record/891104},
}