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@ARTICLE{Demir:891164,
author = {Demir, Fatih and Kizhakkedathu, Jayachandran N. and
Rinschen, Markus M. and Huesgen, Pitter F.},
title = {{MANTI}: {A}utomated {A}nnotation of {P}rotein
{N}-{T}ermini for {R}apid {I}nterpretation of
{N}-{T}erminome {D}ata {S}ets},
journal = {Analytical chemistry},
volume = {93},
number = {13},
issn = {1520-6882},
address = {Columbus, Ohio},
publisher = {American Chemical Society},
reportid = {FZJ-2021-01402},
pages = {5596–5605},
year = {2021},
abstract = {Site-specific proteolytic processing is an important,
irreversible post-translational protein modification with
implications in many diseases. Enrichment of protein
N-terminal peptides followed by mass spectrometry-based
identification and quantification enables proteome-wide
characterization of proteolytic processes and protease
substrates but is challenged by the lack of specific
annotation tools. A common problem is, for example,
ambiguous matches of identified peptides to multiple protein
entries in the databases used for identification. We
developed MaxQuant Advanced N-termini Interpreter (MANTI), a
standalone Perl software with an optional graphical user
interface that validates and annotates N-terminal peptides
identified by database searches with the popular MaxQuant
software package by integrating information from multiple
data sources. MANTI utilizes diverse annotation information
in a multistep decision process to assign a conservative
preferred protein entry for each N-terminal peptide,
enabling automated classification according to the likely
origin and determines significant changes in N-terminal
peptide abundance. Auxiliary R scripts included in the
software package summarize and visualize key aspects of the
data. To showcase the utility of MANTI, we generated two
large-scale TAILS N-terminome data sets from two different
animal models of chemically and genetically induced kidney
disease, puromycin adenonucleoside-treated rats (PAN), and
heterozygous Wilms Tumor protein 1 mice (WT1). MANTI enabled
rapid validation and autonomous annotation of >10 000
identified terminal peptides, revealing novel proteolytic
proteoforms in 905 and 644 proteins, respectively.
Quantitative analysis indicated that proteolytic activities
with similar sequence specificity are involved in the
pathogenesis of kidney injury and proteinuria in both
models, whereas coagulation processes and complement
activation were specifically induced after chemical injury.},
cin = {ZEA-3},
ddc = {540},
cid = {I:(DE-Juel1)ZEA-3-20090406},
pnm = {217 - Für eine nachhaltige Bio-Ökonomie – von
Ressourcen zu Produkten (POF4-217) / ProPlantStress -
Proteolytic processing in plant stress signal transduction
and responses to abiotic stress and pathogen attack
(639905)},
pid = {G:(DE-HGF)POF4-217 / G:(EU-Grant)639905},
typ = {PUB:(DE-HGF)16},
pubmed = {33729755},
UT = {WOS:000638986400032},
doi = {10.1021/acs.analchem.1c00310},
url = {https://juser.fz-juelich.de/record/891164},
}