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@ARTICLE{Janouschek:893389,
      author       = {Janouschek, Hildegard and Chase, Henry W. and Sharkey,
                      Rachel J. and Peterson, Zeru J. and Camilleri, Julia and
                      Abel, Ted and Eickhoff, Simon B. and Nickl-Jockschat,
                      Thomas},
      title        = {{T}he functional neural architecture of dysfunctional
                      reward processing in autism},
      journal      = {NeuroImage: Clinical},
      volume       = {31},
      issn         = {2213-1582},
      address      = {[Amsterdam u.a.]},
      publisher    = {Elsevier},
      reportid     = {FZJ-2021-02730},
      pages        = {102700 -},
      year         = {2021},
      abstract     = {Functional imaging studies have found differential neural
                      activation patterns during reward-paradigms in patients with
                      autism spectrum disorder (ASD) compared to neurotypical
                      controls. However, publications report conflicting results
                      on the directionality and location of these aberrant
                      activations. We here quantitatively summarized relevant fMRI
                      papers in the field using the anatomical likelihood
                      estimation (ALE) algorithm. Patients with ASD consistently
                      showed hypoactivations in the striatum across studies,
                      mainly in the right putamen and accumbens. These regions are
                      functionally involved in the processing of rewards and are
                      enrolled in extensive neural networks involving limbic,
                      cortical, thalamic and mesencephalic regions. The striatal
                      hypo-activations found in our ALE meta-analysis, which
                      pooled over contrasts derived from the included studies on
                      reward-processing in ASD, highlight the role of the striatum
                      as a key neural correlate of impaired reward processing in
                      autism. These changes were present for studies using social
                      and non-social stimuli alike. The involvement of these
                      regions in extensive networks associated with the processing
                      of both positive and negative emotion alike might hint at
                      broader impairments of emotion processing in the disorder.},
      cin          = {INM-7},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-7-20090406},
      pnm          = {5251 - Multilevel Brain Organization and Variability
                      (POF4-525)},
      pid          = {G:(DE-HGF)POF4-5251},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {34161918},
      UT           = {WOS:000689516300001},
      doi          = {10.1016/j.nicl.2021.102700},
      url          = {https://juser.fz-juelich.de/record/893389},
}