% IMPORTANT: The following is UTF-8 encoded.  This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.

@ARTICLE{Schweitzer:902213,
      author       = {Schweitzer, Finja and Goereci, Yasemin and Franke,
                      Christiana and Silling, Steffi and Bösl, Fabian and Maier,
                      Franziska and Heger, Eva and Deiman, Birgit and Prüss,
                      Harald and Onur, Oezguer A. and Klein, Florian and Fink,
                      Gereon Rudolf and Di Cristanziano, Veronica and Warnke,
                      Clemens},
      title        = {{C}erebrospinal {F}luid {A}nalysis {P}ost–{COVID}-19 {I}s
                      {N}ot {S}uggestive of {P}ersistent {C}entral {N}ervous
                      {S}ystem {I}nfection},
      journal      = {Annals of neurology},
      volume       = {91},
      number       = {1},
      issn         = {1531-8249},
      address      = {Hoboken, NJ},
      publisher    = {Wiley-Blackwell},
      reportid     = {FZJ-2021-04101},
      pages        = {150-157},
      year         = {2022},
      abstract     = {This study was undertaken to assess whether SARS-CoV-2
                      causes a persistent central nervous system infection.
                      SARS-CoV-2–specific antibody index and SARS-CoV-2 RNA were
                      studied in cerebrospinal fluid following COVID-19.
                      Cerebrospinal fluid was assessed between days 1 and 30 (n =
                      12), between days 31 and 90 (n = 8), or later than 90 days
                      (post–COVID-19, n = 20) after COVID-19 diagnosis.
                      SARS-CoV-2 RNA was absent in all patients, and in none of
                      the 20 patients with post–COVID-19 syndrome were
                      intrathecally produced anti–SARS-CoV-2 antibodies
                      detected. The absence of evidence of SARS-CoV-2 in
                      cerebrospinal fluid argues against a persistent central
                      nervous system infection as a cause of neurological or
                      neuropsychiatric post–COVID-19 syndrome. ANN NEUROL
                      2022;91:150–157},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {5251 - Multilevel Brain Organization and Variability
                      (POF4-525)},
      pid          = {G:(DE-HGF)POF4-5251},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:34724243},
      UT           = {WOS:000720889800001},
      doi          = {10.1002/ana.26262},
      url          = {https://juser.fz-juelich.de/record/902213},
}