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@INPROCEEDINGS{Tan:902786,
author = {Tan, Zihan and Calandrini, Vania and Dhont, Jan K.G. and
Winkler, Roland G. and Naegele, Gerhard},
title = {{Q}uasi-two-dimensional diffusion of interacting protein
monomers and dimers: {A} {MPC} simulation study},
reportid = {FZJ-2021-04557},
year = {2021},
note = {References:[1] G. Gompper, T. Ihle, D. M. Kroll, R. G.
Winkler, Adv. Polym. Sci, 221, 1-87 (2008). [2] Z. Tan, J.
K. G. Dhont, V. Calandrini, and G. Nägele, paper in
preparation.[3] S. Panzuela and R. Delgado-Buscalioni, Phys.
Rev. Lett., 121, 048101 (2018).[4] Z. Tan, V. Calandrini, J.
K. G. Dhont, G. Nägele, and R. G. Winkler, arXiv:2105.01492
(2021).},
abstract = {Understanding lateral diffusion of proteins along a
membrane is of importance in biological soft matter science.
An example in case is postsynaptic neuronal signal
transduction where specific proteins diffuse alongside a
postsynaptic membrane, triggering a cascade of biochemical
processes. There are challenging questions to answer such as
how the collective and self-diffusion of the proteins are
affected by their direct and hydrodynamic interactions for
larger areal protein concentrations. Using the
multi-particle collision dynamics (MPC) simulation methods
[1], we explore protein diffusion under
quasi-two-dimensional (Q2D) confinement, for two different
model systems of proteins. In the first system, the proteins
are modeled as Brownian spheres interacting, respectively,
by a hard-sphere potential serving as a reference potential,
and by a soft potential with competing short-range
attractive and long-range repulsive parts. For a
minimalistic description of proteins diffusing along a
cytosol-membrane interface, the Brownian spheres are
confined to lateral motion in a planar monolayer embedded in
an unbound three-dimensional Newtonian fluid. The time
scales in the dynamic simulations extend from very short
times where inertial effects are resolved, up to long times
where the solvent-mediated hydrodynamic interactions between
the proteins are fully developed and non-retarded [2]. By
computing velocity autocorrelation functions, mean-square
displacements and Fourier-space current auto-correlation
functions, we quantify how concentration-induced
correlations affect, e.g., the anomalous enhancement of
large-scale collective diffusion under Q2D confinement [3],
and the development of inter-protein hydrodynamic
interactions by multiple scattering of sound and by
vorticity diffusion [2]. The second model system relates to
the diffusion of a human dumbbell-shaped M2 muscarinic
acetylcholine receptor protein where the upper segment is
embedded in the neuronal cell membrane, and the lower one in
the cytosol. The protein is simply modelled by a two-beads
dimer with the upper bead immersed in a high-viscosity fluid
sheet (fluid A) mimicking the membrane, and the lower one in
a lower-viscosity fluid B mimicking the intra- and also
extracellular environment. We use a recently developed MPC
scheme for generating a fluid sheet A inside another fluid B
[4]. Using this mesoscale method, diffusion can be probed
over time spans not accessible in atomistic MD simulations
of proteins. We study the mean squared displacement and
velocity autocorrelation function of the individual bead
centres and the hydrodynamic centre of mobility of the
dumbbell, in dependence of the viscosity ratio sheet
thickness and interfacial bead distances.},
month = {Jul},
date = {2021-07-19},
organization = {11th LIQUID MATTER CONFERENCE
2020/2021, Prague/Online (Czech
Republic), 19 Jul 2021 - 23 Jul 2021},
subtyp = {After Call},
cin = {IBI-4 / IAS-5 / IBI-5 / INM-9},
cid = {I:(DE-Juel1)IBI-4-20200312 / I:(DE-Juel1)IAS-5-20120330 /
I:(DE-Juel1)IBI-5-20200312 / I:(DE-Juel1)INM-9-20140121},
pnm = {5244 - Information Processing in Neuronal Networks
(POF4-524)},
pid = {G:(DE-HGF)POF4-5244},
typ = {PUB:(DE-HGF)24},
url = {https://juser.fz-juelich.de/record/902786},
}