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@ARTICLE{Bogorodskiy:903605,
author = {Bogorodskiy, Andrey and Okhrimenko, Ivan and Burkatovskii,
Dmitrii and Jakobs, Philipp and Maslov, Ivan and Gordeliy,
Valentin and Dencher, Norbert A. and Gensch, Thomas and
Voos, Wolfgang and Altschmied, Joachim and Haendeler, Judith
and Borshchevskiy, Valentin},
title = {{R}ole of {M}itochondrial {P}rotein {I}mport in
{A}ge-{R}elated {N}eurodegenerative and {C}ardiovascular
{D}iseases},
journal = {Cells},
volume = {10},
number = {12},
issn = {2073-4409},
address = {Basel},
publisher = {MDPI},
reportid = {FZJ-2021-05259},
pages = {3528 -},
year = {2021},
abstract = {Mitochondria play a critical role in providing energy,
maintaining cellular metabolism, and regulating cell
survival and death. To carry out these crucial functions,
mitochondria employ more than 1500 proteins, distributed
between two membranes and two aqueous compartments. An
extensive network of dedicated proteins is engaged in
importing and sorting these nuclear-encoded proteins into
their designated mitochondrial compartments. Defects in this
fundamental system are related to a variety of pathologies,
particularly engaging the most energy-demanding tissues. In
this review, we summarize the state-of-the-art knowledge
about the mitochondrial protein import machinery and
describe the known interrelation of its failure with
age-related neurodegenerative and cardiovascular diseases.},
cin = {IBI-1},
ddc = {570},
cid = {I:(DE-Juel1)IBI-1-20200312},
pnm = {5243 - Information Processing in Distributed Systems
(POF4-524)},
pid = {G:(DE-HGF)POF4-5243},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34944035},
UT = {WOS:000741498500001},
doi = {10.3390/cells10123528},
url = {https://juser.fz-juelich.de/record/903605},
}