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@ARTICLE{Park:904412,
author = {Park, Shinwon and Haak, Koen V. and Cho, Han Byul and Valk,
Sofie L. and Bethlehem, Richard A. I. and Milham, Michael P.
and Bernhardt, Boris C. and Di Martino, Adriana and Hong,
Seok-Jun},
title = {{A}typical {I}ntegration of {S}ensory-to-{T}ransmodal
{F}unctional {S}ystems {M}ediates {S}ymptom {S}everity in
{A}utism},
journal = {Frontiers in psychiatry},
volume = {12},
issn = {1664-0640},
address = {Lausanne},
publisher = {Frontiers Research Foundation},
reportid = {FZJ-2021-05982},
pages = {699813},
year = {2021},
abstract = {A notable characteristic of autism spectrum disorder (ASD)
is co-occurring deficits in low-level sensory processing and
high-order social interaction. While there is evidence
indicating detrimental cascading effects of sensory
anomalies on the high-order cognitive functions in ASD, the
exact pathological mechanism underlying their atypical
functional interaction across the cortical hierarchy has not
been systematically investigated. To address this gap, here
we assessed the functional organisation of sensory and motor
areas in ASD, and their relationship with subcortical and
high-order trandmodal systems. In a resting-state fMRI data
of 107 ASD and 113 neurotypical individuals, we applied
advanced connectopic mapping to probe functional
organization of primary sensory/motor areas, together with
targeted seed-based intrinsic functional connectivity (iFC)
analyses. In ASD, the connectopic mapping revealed
topological anomalies (i.e., excessively more segregated
iFC) in the motor and visual areas, the former of which
patterns showed association with the symptom severity of
restricted and repetitive behaviors. Moreover, the
seed-based analysis found diverging patterns of ASD-related
connectopathies: decreased iFCs within the sensory/motor
areas but increased iFCs between sensory and subcortical
structures. While decreased iFCs were also found within the
higher-order functional systems, the overall proportion of
this anomaly tends to increase along the level of cortical
hierarchy, suggesting more dysconnectivity in the
higher-order functional networks. Finally, we demonstrated
that the association between low-level sensory/motor iFCs
and clinical symptoms in ASD was mediated by the high-order
transmodal systems, suggesting pathogenic functional
interactions along the cortical hierarchy. Findings were
largely replicated in the independent dataset. These results
highlight that atypical integration of sensory-to-high-order
systems contributes to the complex ASD symptomatology.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5251 - Multilevel Brain Organization and Variability
(POF4-525)},
pid = {G:(DE-HGF)POF4-5251},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34489757},
UT = {WOS:000696038500001},
doi = {10.3389/fpsyt.2021.699813},
url = {https://juser.fz-juelich.de/record/904412},
}