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@ARTICLE{Puhlmann:904413,
author = {Puhlmann, L. M. C. and Linz, R. and Valk, S. L. and
Vrticka, P. and Vos de Wael, R. and Bernasconi, A. and
Bernasconi, N. and Caldairou, B. and Papassotiriou, I. and
Chrousos, G. P. and Bernhardt, B. C. and Singer, T. and
Engert, V.},
title = {{A}ssociation between hippocampal structure and serum
{B}rain-{D}erived {N}eurotrophic {F}actor ({BDNF}) in
healthy adults: {A} registered report},
journal = {NeuroImage},
volume = {236},
issn = {1053-8119},
address = {Orlando, Fla.},
publisher = {Academic Press},
reportid = {FZJ-2021-05983},
pages = {118011 -},
year = {2021},
abstract = {The hippocampus is a highly plastic brain structure
supporting functions central to human cognition.
Morphological changes in the hippocampus have been
implicated in development, aging, as well as in a broad
range of neurological and psychiatric disorders. A growing
body of research suggests that hippocampal plasticity is
closely linked to the actions of brain-derived neurotrophic
factor (BDNF). However, evidence on the relationship between
hippocampal volume (HCV) and peripheral BDNF levels is
scarce and limited to elderly and patient populations.
Further, despite evidence that BDNF expression differs
throughout the hippocampus and is implicated in adult
neurogenesis specifically in the dentate gyrus, no study has
so far related peripheral BDNF levels to the volumes of
individual hippocampal subfields. Besides its clinical
implications, BDNF-facilitated hippocampal plasticity plays
an important role in regulating cognitive and affective
processes. In the current registered report, we investigated
how serum BDNF (sBDNF) levels relate to volumes of the
hippocampal formation and its subfields in a large sample of
healthy adults (N = 279, 160 f) with a broad age range
(20–55 years, mean 40.5) recruited in the context of the
ReSource Project. We related HCV to basal sBDNF and, in a
subsample (n = 103, 57 f), to acute stress-reactive change
in sBDNF. We further tested the role of age as a moderator
of both associations. Contrary to our hypotheses, neither
basal sBDNF levels nor stress-reactive sBDNF change were
associated with total HCV or volume of the dentate
gyrus/cornu ammonis 4 (DG/CA4) subfield. We also found no
evidence for a moderating effect of age on any of these
associations. Our null results provide a first point of
reference on the relationship between sBDNF and HCV in
healthy mid-age, in contrast to patient or aging
populations. We suggest that sBDNF levels have limited
predictive value for morphological differences of the
hippocampal structure when notable challenge to its neuronal
integrity or to neurotrophic capacity is absent.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5251 - Multilevel Brain Organization and Variability
(POF4-525)},
pid = {G:(DE-HGF)POF4-5251},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:33852941},
UT = {WOS:000670278100001},
doi = {10.1016/j.neuroimage.2021.118011},
url = {https://juser.fz-juelich.de/record/904413},
}