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@INPROCEEDINGS{Foerges:910591,
author = {Foerges, Anna Linea and Elmenhorst, Eva-Maria and Lange,
Denise and Baur, Diego and Beer, Simone and Kroll, Tina and
Neumaier, Bernd and Aeschbach, Daniel and Bauer, Andreas and
Landolt, Hans-Peter and Elmenhorst, David},
title = {{D}oes repeated coffee consumption during chronic sleep
restriction affect {A}1 adenosine receptor availability in
humans?},
reportid = {FZJ-2022-03969},
year = {2022},
abstract = {Objectives:Adenosine and cerebral adenosine A1 (A1AR) and
A2A receptors are important modulators of the sleep-wake
cycle and regulate to some extent the sleep-wake
homeostasis. Acute sleep deprivation has been shown to
result in an upregulation of A1ARs in human and rat brains.
The stimulating effects of caffeine are evoked through
non-selective antagonism at adenosine receptors. Using
positron emission tomography (PET), we investigate the
effect of repeated coffee consumption during chronic sleep
restriction and subsequent coffee abstinence after recovery
sleep on cerebral A1AR availability and occupancy in
humans.Methods:Thirty healthy volunteers (28±5 years, 15f)
completed an in-lab study including three [18F]CPFPX PET
scans to determine cerebral A1AR availability after
subsequent exposure to rested (3 nights with 8h time in bed
(TIB)), chronically sleep restricted (5 nights with 5h TIB),
and recovery (one night with 8h TIB) conditions.
Participants either consumed freshly brewed coffee (n = 17)
or decaffeinated coffee (n = 13) during 5 days of sleep
restriction (prior caffeine abstinence > 10 days). Regular
coffee contained 200 mg caffeine at 7.30 a.m. and 100 mg
caffeine at 2.00 p.m., decaffeinated coffee contained 4 mg
and 2 mg, respectively. PET scans were conducted at the same
time of day under caffeine-abstinent rested conditions,
roughly 7h after the latest coffee intake after sleep
restriction, and after ~ 31h of coffee abstention after
recovery. Caffeine levels in saliva were determined
repeatedly. Cerebral A1AR availability was quantified by
distribution volume (VT) and occupancy levels were
calculated by applying the Lassen plot including cortical
and subcortical areas, cerebellum, and pons.Results:In the
decaffeinated coffee group, no differences in cerebral A1AR
availability were found between baseline condition, 5 days
of sleep restriction and one night of recovery sleep.
Repeated administration of regular coffee resulted in a
displacement of [18F]CPFPX binding of 19 ± 13 $\%$ on
average. One day after coffee abstention and recovery sleep,
VT values did not differ from baseline.Conclusions:Our data
suggest that neither chronic sleep restriction for 5 days
nor combination with repeated caffeine consumption result in
a persistent change in the regulation of cerebral A1AR
availability.Financial support:The work was supported by the
Institute for Scientific Information on Coffee, the Swiss
National Science Foundation (# $320030_163439)$ and
respective institutional funds.},
month = {Sep},
date = {2022-09-27},
organization = {The 26th Conference of the European
Sleep Research Society, Athens
(Greece), 27 Sep 2022 - 30 Sep 2022},
subtyp = {After Call},
cin = {INM-2 / INM-5},
cid = {I:(DE-Juel1)INM-2-20090406 / I:(DE-Juel1)INM-5-20090406},
pnm = {5253 - Neuroimaging (POF4-525)},
pid = {G:(DE-HGF)POF4-5253},
typ = {PUB:(DE-HGF)24},
url = {https://juser.fz-juelich.de/record/910591},
}