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@ARTICLE{Choi:910729,
author = {Choi, Hyoungshin and Byeon, Kyoungseob and Park, Bo-yong
and Lee, Jong-eun and Valk, Sofie and Bernhardt, Boris and
Martino, Adriana Di and Milham, Michael and Hong, Seok-Jun
and Park, Hyunjin},
title = {{D}iagnosis-informed connectivity subtyping discovers
subgroups of autism with reproducible symptom profiles},
journal = {NeuroImage},
volume = {256},
issn = {1053-8119},
address = {Orlando, Fla.},
publisher = {Academic Press},
reportid = {FZJ-2022-04099},
pages = {119212 -},
year = {2022},
abstract = {Clinical heterogeneity has been one of the main barriers to
develop effective biomarkers and therapeutic strategies in
autism spectrum disorder (ASD). Recognizing this challenge,
much effort has been made in recent neuroimaging studies to
find biologically more homogeneous subgroups (called
‘neurosubtypes’) in autism. However, most approaches
have rarely evaluated how much the employed features in
subtyping represent the core anomalies of ASD, obscuring its
utility in actual clinical diagnosis. To address this, we
combined two data-driven methods, ‘connectome-based
gradient’ and ‘functional random forest’, collectively
allowing to discover reproducible neurosubtypes based on
resting-state functional connectivity profiles that are
specific to ASD. Indeed, the former technique provides the
features (as input for subtyping) that effectively summarize
whole-brain connectome variations in both normal and ASD
conditions, while the latter leverages a supervised random
forest algorithm to inform diagnostic labels to clustering,
which makes neurosubtyping driven by the features of ASD
core anomalies. Applying this framework to the open-sharing
Autism Brain Imaging Data Exchange repository data
(discovery, n = 103/108 for ASD/typically developing [TD];
replication, n = 44/42 for ASD/TD), we found three dominant
subtypes of functional gradients in ASD and three subtypes
in TD. The subtypes in ASD revealed distinct connectome
profiles in multiple brain areas, which are associated with
different Neurosynth-derived cognitive functions previously
implicated in autism studies. Moreover, these subtypes
showed different symptom severity, which degree co-varies
with the extent of functional gradient changes observed
across the groups. The subtypes in the discovery and
replication datasets showed similar symptom profiles in
social interaction and communication domains, confirming a
largely reproducible brain-behavior relationship. Finally,
the connectome gradients in ASD subtypes present both common
and distinct patterns compared to those in TD, reflecting
their potential overlap and divergence in terms of
developmental mechanisms involved in the manifestation of
large-scale functional networks. Our study demonstrated a
potential of the diagnosis-informed subtyping approach in
developing a clinically useful brain-based classification
system for future ASD research.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {35430361},
UT = {WOS:000830364700005},
doi = {10.1016/j.neuroimage.2022.119212},
url = {https://juser.fz-juelich.de/record/910729},
}