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@ARTICLE{Hemmer:917514,
author = {Hemmer, Stefanie and Schulte, Marianne and
Knieps-Grünhagen, Esther and Granzin, Joachim and Willbold,
Dieter and Jaeger, Karl-Erich and Batra-Safferling, Renu and
Panwalkar, Vineet and Krauss, Ulrich},
title = {{R}esidue alterations within a conserved hydrophobic pocket
influence light, oxygen, voltage photoreceptor dark
recovery},
journal = {Photochemical $\&$ photobiological sciences},
volume = {22},
issn = {1474-905X},
address = {Heidelberg},
publisher = {Springer},
reportid = {FZJ-2023-00727},
pages = {713-727},
year = {2023},
abstract = {Light, oxygen, voltage (LOV) photoreceptors are widely
distributed throughout all kingdoms of life, and have in
recent years, due to their modular nature, been broadly used
as sensor domains for the construction of optogenetic tools.
For understanding photoreceptor function as well as for
optogenetic tool design and fine-tuning, a detailed
knowledge of the photophysics, photochemistry, and
structural changes underlying the LOV signaling paradigm is
instrumental. Mutations that alter the lifetime of the
photo-adduct signaling state represent a convenient handle
to tune LOV sensor on/off kinetics and, thus, steady-state
on/off equilibria of the photoreceptor (or optogenetic
switch). Such mutations, however, should ideally only
influence sensor kinetics, while being benign with regard to
the nature of the structural changes that are induced by
illumination, i.e., they should not result in a disruption
of signal transduction. In the present study, we identify a
conserved hydrophobic pocket for which mutations have a
strong impact on the adduct-state lifetime across different
LOV photoreceptor families. Using the slow cycling bacterial
short LOV photoreceptor PpSB1-LOV, we show that the I48T
mutation within this pocket, which accelerates adduct
rupture, is otherwise structurally and mechanistically
benign, i.e., light-induced structural changes, as probed by
NMR spectroscopy and X-ray crystallography, are not altered
in the variant. Additional mutations within the pocket of
PpSB1-LOV and the introduction of homologous mutations in
the LOV photoreceptor YtvA of Bacillus subtilis and the
Avena sativa LOV2 domain result in similarly altered
kinetics. Given the conserved nature of the corresponding
structural region, the here identified mutations should find
application in dark-recovery tuning of optogenetic tools and
LOV photoreceptors, alike.},
cin = {IBG-1 / IMET / IBI-7},
ddc = {620},
cid = {I:(DE-Juel1)IBG-1-20101118 / I:(DE-Juel1)IMET-20090612 /
I:(DE-Juel1)IBI-7-20200312},
pnm = {5241 - Molecular Information Processing in Cellular Systems
(POF4-524) / 2171 - Biological and environmental resources
for sustainable use (POF4-217)},
pid = {G:(DE-HGF)POF4-5241 / G:(DE-HGF)POF4-2171},
typ = {PUB:(DE-HGF)16},
pubmed = {36480084},
UT = {WOS:000895631300002},
doi = {10.1007/s43630-022-00346-5},
url = {https://juser.fz-juelich.de/record/917514},
}