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@ARTICLE{EriaOliveira:1025966,
author = {Eria-Oliveira, Ana-Sofia and Folacci, Mathilde and Chassot,
Anne Amandine and Fedou, Sandrine and Thézé, Nadine and
Zabelskii, Dmitrii and Alekseev, Alexey and Bamberg, Ernst
and Gordeliy, Valentin and Sandoz, Guillaume and Vivaudou,
Michel},
title = {{H}ijacking of internal calcium dynamics by intracellularly
residing viral rhodopsins},
journal = {Nature Communications},
volume = {15},
number = {1},
issn = {2041-1723},
address = {[London]},
publisher = {Nature Publishing Group UK},
reportid = {FZJ-2024-03244},
pages = {65},
year = {2024},
abstract = {Rhodopsins are ubiquitous light-driven membrane proteins
with diverse functions, including ion transport. Widely
distributed, they are also coded in the genomes of giant
viruses infecting phytoplankton where their function is not
settled. Here, we examine the properties of OLPVR1 (Organic
Lake Phycodnavirus Rhodopsin) and two other type 1 viral
channelrhodopsins (VCR1s), and demonstrate that VCR1s
accumulate exclusively intracellularly, and, upon
illumination, induce calcium release from intracellular
IP3-dependent stores. In vivo, this light-induced calcium
release is sufficient to remote control muscle contraction
in VCR1-expressing tadpoles. VCR1s natively confer
light-induced Ca2+ release, suggesting a distinct mechanism
for reshaping the response to light of virus-infected algae.
The ability of VCR1s to photorelease calcium without
altering plasma membrane electrical properties marks them as
potential precursors for optogenetics tools, with potential
applications in basic research and medicine.},
cin = {IBI-7},
ddc = {500},
cid = {I:(DE-Juel1)IBI-7-20200312},
pnm = {5241 - Molecular Information Processing in Cellular Systems
(POF4-524)},
pid = {G:(DE-HGF)POF4-5241},
typ = {PUB:(DE-HGF)16},
pubmed = {38167346},
UT = {WOS:001288614600035},
doi = {10.1038/s41467-023-44548-6},
url = {https://juser.fz-juelich.de/record/1025966},
}