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@ARTICLE{Keller:1052340,
author = {Keller, Lara and Lotter, Leon D. and Eickhoff, Claudia R.
and Eickhoff, Simon B. and Otten, Katharina and
Herpertz-Dahlmann, Beate and Seitz, Jochen},
title = {{S}tructural brain alterations in anorexia nervosa: {A}
global brain volume and anatomical likelihood estimation
({ALE}) meta-analysis combined with a functional decoding
approach},
journal = {NeuroImage: Clinical},
volume = {na},
issn = {2213-1582},
address = {[Amsterdam u.a.]},
publisher = {Elsevier},
reportid = {FZJ-2026-00943},
pages = {103950},
year = {2026},
abstract = {Substantial brain volume loss is well-documented during
acute anorexia nervosa (AN); however, longitudinal outcomes
are unclear. Our comprehensive meta-analysis investigated
global and regional structural brain alterations in adult
and adolescent individuals with AN by extracting reported
brain volume scores and neuroimaging coordinates from the
literature. Results showed significant global brain volume
reductions in gray matter (GM), white matter (WM), and
increases in cerebrospinal fluid (CSF) in acute AN (N = 1130
patients; N = 40 papers), gradually improving upon weight
rehabilitation. However, even after 1.5 years of recovery,
significantly lower global GM volume compared to healthy
controls was found (N = 232 patients; N = 12 papers).
Regarding potential regional changes, our search identified
35 eligible papers with neuroimaging coordinates for 412
foci as input for our anatomical likelihood estimation (ALE)
analyses. The results revealed widespread reductions of GM
volume and cortical thickness, but notably also identified
consistently affected brain regions including the cingulate
gyrus, precentral gyrus, and precuneus. Spatial
colocalization analyses using the Neurosynth data base
indicated brain areas associated with eating, food, threat,
and reinforcement to be relatively preserved. The findings
of our meta-analysis contribute to a better understanding of
the underlying pathophysiology of AN, the time course and
residuals of brain structural alterations during recovery
and clinical implications potentially relevant for
more-targeted treatment options.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
doi = {10.1016/j.nicl.2026.103950},
url = {https://juser.fz-juelich.de/record/1052340},
}