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@ARTICLE{Timmermann:201196,
      author       = {Timmermann, L. and Eggers, C. and Salimi Dafsari, H. and
                      Pauls, K. and Barbe, M.},
      title        = {{M}orbus {P}arkinson: {A}ktuelle {S}tandards in
                      {D}iagnostik und {T}herapie},
      journal      = {Fortschritte der Neurologie, Psychiatrie},
      volume       = {80},
      number       = {10},
      issn         = {1439-3522},
      address      = {Stuttgart [u.a.]},
      publisher    = {Thieme},
      reportid     = {FZJ-2015-03501},
      pages        = {560 - 569},
      year         = {2012},
      abstract     = {Idiopathic Parkinson’s disease is still a clinical
                      diagnosis. However, modern imaging and nuclear techniques
                      allow very early diagnosis and lead to higher security in
                      the differential diagnosis between idiopathic Parkinson’s
                      disease and atypical Parkinson syndromes. At early stages of
                      the disease, modification of disease progression and symptom
                      control are key factors of the therapy. Continuous
                      dopaminergic stimulation is even more important at later
                      stages with first fluctuations. In stages where conservative
                      medical options have been exhausted continuous pump
                      therapies with Duodopa and apomorphine are attractive
                      options. Deep brain stimulation in the subthalamic nucleus
                      has turned out in the last years, especially in younger
                      patients, to be a highly successful treatment option. Deep
                      drain stimulation requires, however, a close preoperative
                      work-up and individual consideration of potential effects
                      and side effects.},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {333 - Pathophysiological Mechanisms of Neurological and
                      Psychiatric Diseases (POF2-333)},
      pid          = {G:(DE-HGF)POF2-333},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000310409200002},
      pubmed       = {pmid:23033202},
      doi          = {10.1055/s-0032-1312738},
      url          = {https://juser.fz-juelich.de/record/201196},
}