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@ARTICLE{SchulteRther:22510,
      author       = {Schulte-Rüther, M. and Mainz, V. and Fink, G.R. and
                      Herpertz-Dahlmann, B. and Konrad, K.},
      title        = {{T}heory of mind and the brain in anorexia nervosa:
                      relation to treatment outcome.},
      journal      = {Journal of the American Academy of Child and Adolescent
                      Psychiatry},
      volume       = {51},
      issn         = {0890-8567},
      address      = {Kidlington [u.a.]},
      publisher    = {Elsevier},
      reportid     = {PreJuSER-22510},
      pages        = {832 - 841},
      year         = {2012},
      note         = {This study was supported by the Bundesministerium fur
                      Bildung und Forschung grant BMBF 01GV0602 (B.H.-D.,
                      K.K.).Dr. Fink has served as an editorial board member of
                      Cortex, Zeitschrift fur Neuropsychologie and Fortschritte
                      der Neurologie Psychiatrie. He has received royalties from
                      the publication of the book 'Funktionelle MRT in Psychiotrie
                      und Neurologie" and "Neurologische Differentialdiagnose,"
                      has received honoraria for speaking engagements from Teva,
                      GlaxoSmithKline, and Boehringer Ingelheim, and has received
                      research support from the Bundesministerium fur Bildung und
                      Forschung and the Deutsche Forschungsgemeinschaft. Dr.
                      Herpertz-Dohlmann has served as a consultant to Eli Lilly
                      and Co. and has received industry research funding from
                      Medice and Vifor. Dr. Konrad has served as an editorial
                      board member of the Journal of Neural Transmission, the
                      Journal of Child Psychology, and Zeitschrift fur Kinder-und
                      Jugendpsychiatrie, has received speaking fees from Eli Lilly
                      and Co., Novartis, and Medice, has received industry
                      research funding from Vifor, and has received research
                      support from the Bundesministerium fur Bildung und Forschung
                      and the Deutsche Forschungsgemeinschaft. Drs. Schulte-Ruther
                      and Mainz report no biomedical financial interests or
                      potential conflicts of interest.},
      abstract     = {Converging evidence suggests deficits in theory-of-mind
                      (ToM) processing in patients with anorexia nervosa (AN). The
                      present study aimed at elucidating the neural mechanisms
                      underlying ToM-deficits in AN.A total of 19 adolescent
                      patients with AN and 21 age-matched controls were
                      investigated using functional magnetic resonance imaging
                      during performance of a ToM-task at two time points
                      (in-patients: admission to hospital and discharge after
                      weight recovery). Clinical outcomes in patients were
                      determined 1 year after admission.Irrespective of the time
                      point, AN patients showed reduced activation in middle and
                      anterior temporal cortex and in the medial prefrontal
                      cortex. Hypoactivation in the medial prefrontal cortex at
                      admission to hospital (T1) was correlated with clinical
                      outcome at follow-up.Hypoactivation in the brain network
                      supporting theory of mind may be associated with a
                      social-cognitive endophenotype reflecting impairments of
                      social functioning in anorexia nervosa which is predictive
                      for a poor outcome at 1-year follow-up.},
      keywords     = {J (WoSType)},
      cin          = {INM-3},
      ddc          = {150},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {Funktion und Dysfunktion des Nervensystems (FUEK409) /
                      89571 - Connectivity and Activity (POF2-89571)},
      pid          = {G:(DE-Juel1)FUEK409 / G:(DE-HGF)POF2-89571},
      shelfmark    = {Psychology, Developmental / Pediatrics / Psychiatry},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:22840554},
      UT           = {WOS:000307128300011},
      doi          = {10.1016/j.jaac.2012.06.007},
      url          = {https://juser.fz-juelich.de/record/22510},
}