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@ARTICLE{Rose:828247,
author = {Rose, Michael and Kloten, Vera and Noetzel, Erik and Gola,
Lukas and Ehling, Josef and Heide, Timon and Meurer, Steffen
K. and Gaiko-Shcherbak, Aljona and Sechi, Antonio S. and
Huth, Sebastian and Weiskirchen, Ralf and Klaas, Oliver and
Antonopoulos, Wiebke and Lin, Qiong and Wagner, Wolfgang and
Veeck, Jürgen and Gremse, Felix and Steitz, Julia and
Knüchel, Ruth and Dahl, Edgar},
title = {{ITIH}5 mediates epigenetic reprogramming of breast cancer
cells},
journal = {Molecular cancer},
volume = {16},
number = {1},
issn = {1476-4598},
address = {London},
publisher = {Biomed Central},
reportid = {FZJ-2017-02215},
pages = {44},
year = {2017},
abstract = {Extracellular matrix (ECM) is known to maintain epithelial
integrity. In carcinogenesis ECM degradation triggers
metastasis by controlling migration and differentiation
including cancer stem cell (CSC) characteristics. The
ECM-modulator inter- α-trypsin inhibitor heavy chain family
member five (ITIH5) was recently identified as tumor
suppressor potentially involved in impairing breast cancer
progression but molecular mechanisms underlying its function
are still elusive.},
cin = {ICS-7},
ddc = {610},
cid = {I:(DE-Juel1)ICS-7-20110106},
pnm = {552 - Engineering Cell Function (POF3-552)},
pid = {G:(DE-HGF)POF3-552},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000397799800002},
pubmed = {pmid:28231808},
doi = {10.1186/s12943-017-0610-2},
url = {https://juser.fz-juelich.de/record/828247},
}