Journal Article

FZJ-2017-02215

http://join2-wiki.gsi.de/foswiki/pub/Main/Artwork/join2_logo100x88.png ITIH5 mediates epigenetic reprogramming of breast cancer cells

Rose, M. (Corresponding author) ; Kloten, V. ; Noetzel, E.FZJ* ; Gola, L. ; Ehling, J. ; Heide, T. ; Meurer, S. K. ; Gaiko-Shcherbak, A.FZJ* ; Sechi, A. S. ; Huth, S. ; Weiskirchen, R. ; Klaas, O. ; Antonopoulos, W. ; Lin, Q. ; Wagner, W. ; Veeck, J. ; Gremse, F. ; Steitz, J. ; Knüchel, R. ; Dahl, E. (Corresponding author)

2017
Biomed Central London

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Please use a persistent id in citations: http://hdl.handle.net/2128/13964  doi:10.1186/s12943-017-0610-2

Abstract: Extracellular matrix (ECM) is known to maintain epithelial integrity. In carcinogenesis ECM degradation triggers metastasis by controlling migration and differentiation including cancer stem cell (CSC) characteristics. The ECM-modulator inter- α-trypsin inhibitor heavy chain family member five (ITIH5) was recently identified as tumor suppressor potentially involved in impairing breast cancer progression but molecular mechanisms underlying its function are still elusive.

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Contributing Institute(s):

1. Biomechanik (ICS-7)

Research Program(s):

1. 552 - Engineering Cell Function (POF3-552) (POF3-552)

Appears in the scientific report 2017

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Database coverage:
; ; ; ; BIOSIS Previews ; Current Contents - Life Sciences ; DOAJ Seal ; Ebsco Academic Search ; IF >= 5 ; JCR ; NCBI Molecular Biology Database ; SCOPUS ; Science Citation Index Expanded ; Thomson Reuters Master Journal List ; Web of Science Core Collection

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