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@ARTICLE{Fabris:856429,
author = {Fabris, Gloria and Lucantonio, Alessandro and Hampe, Nico
and Noetzel, Erik and Hoffmann, Bernd and DeSimone, Antonio
and Merkel, Rudolf},
title = {{N}anoscale {T}opography and {P}oroelastic {P}roperties of
{M}odel {T}issue {B}reast {G}land {B}asement {M}embranes},
journal = {Biophysical journal},
volume = {115},
number = {9},
issn = {0006-3495},
address = {Bethesda, Md.},
publisher = {Soc.},
reportid = {FZJ-2018-05828},
pages = {1770-1782},
year = {2018},
abstract = {Basement membranes (BMs) are thin layers of condensed
extracellular matrix proteins serving as permeability
filters, cellular anchoring sites, and barriers against
cancer cell invasion. It is believed that their
biomechanical properties play a crucial role in determining
cellular behavior and response, especially in mechanically
active tissues like breast glands. Despite this, so far,
relatively little attention has been dedicated to their
analysis because of the difficulty of isolating and handling
such thin layers of material. Here, we isolated BMs derived
from MCF10A spheroids—three-dimensional breast gland model
systems mimicking in vitro the most relevant phenotypic
characteristics of human breast lobules—and characterized
them by atomic force microscopy, enhanced resolution
confocal microscopy, and scanning electron microscopy. By
performing atomic force microscopy height-clamp experiments,
we obtained force-relaxation curves that offered the first
biomechanical data on isolated breast gland BMs to our
knowledge. Based on enhanced resolution confocal microscopy
and scanning electron microscopy imaging data, we modeled
the system as a polymer network immersed in liquid and
described it as a poroelastic material. Finite-element
simulations matching the experimental force-relaxation
curves allowed for the first quantification, to our
knowledge, of the bulk and shear moduli of the membrane as
well as its water permeability. These results represent a
first step toward a deeper understanding of the mechanism of
tensional homeostasis regulating mammary gland activity as
well as its disruption during processes of membrane
breaching and metastatic invasion.},
cin = {ICS-7},
ddc = {570},
cid = {I:(DE-Juel1)ICS-7-20110106},
pnm = {552 - Engineering Cell Function (POF3-552) / 553 - Physical
Basis of Diseases (POF3-553)},
pid = {G:(DE-HGF)POF3-552 / G:(DE-HGF)POF3-553},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30322796},
UT = {WOS:000449422100018},
doi = {10.1016/j.bpj.2018.09.020},
url = {https://juser.fz-juelich.de/record/856429},
}