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@ARTICLE{Hensel:885429,
author = {Hensel, Lukas and Tscherpel, Caroline and Freytag, Jana and
Ritter, Stella and Rehme, Anne K and Volz, Lukas J and
Eickhoff, Simon B and Fink, Gereon R and Grefkes, Christian},
title = {{C}onnectivity-{R}elated {R}oles of {C}ontralesional
{B}rain {R}egions for {M}otor {P}erformance {E}arly after
{S}troke},
journal = {Cerebral cortex},
volume = {31},
number = {2},
issn = {1460-2199},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {FZJ-2020-03826},
pages = {993–1007},
year = {2021},
abstract = {Hemiparesis after stroke is associated with increased
neural activity not only in the lesioned but also in the
contralesional hemisphere. While most studies have focused
on the role of contralesional primary motor cortex (M1)
activity for motor performance, data on other areas within
the unaffected hemisphere are scarce, especially early after
stroke. We here combined functional magnetic resonance
imaging (fMRI) and transcranial magnetic stimulation (TMS)
to elucidate the contribution of contralesional M1, dorsal
premotor cortex (dPMC), and anterior intraparietal sulcus
(aIPS) for the stroke-affected hand within the first 10 days
after stroke. We used "online" TMS to interfere with neural
activity at subject-specific fMRI coordinates while
recording 3D movement kinematics. Interfering with aIPS
activity improved tapping performance in patients, but not
healthy controls, suggesting a maladaptive role of this
region early poststroke. Analyzing effective connectivity
parameters using a Lasso prediction model revealed that
behavioral TMS effects were predicted by the coupling of the
stimulated aIPS with dPMC and ipsilesional M1. In
conclusion, we found a strong link between patterns of
frontoparietal connectivity and TMS effects, indicating a
detrimental influence of the contralesional aIPS on motor
performance early after stroke.Keywords: DCM; diaschisis;
kinematics; motor network; recovery.},
cin = {INM-7 / INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406 / I:(DE-Juel1)INM-3-20090406},
pnm = {571 - Connectivity and Activity (POF3-571) / 5252 - Brain
Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF3-571 / G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {32995880},
UT = {WOS:000646868100019},
doi = {10.1093/cercor/bhaa270},
url = {https://juser.fz-juelich.de/record/885429},
}