% IMPORTANT: The following is UTF-8 encoded.  This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.

@ARTICLE{Borlinghaus:890800,
      author       = {Borlinghaus, Jan and Bolger, Anthony and Schier, Christina
                      and Vogel, Alexander and Usadel, Björn and Gruhlke, Martin
                      CH and Slusarenko, Alan J},
      title        = {{G}enetic and molecular characterization of multicomponent
                      resistance of {P}seudomonas against allicin},
      journal      = {Life science alliance},
      volume       = {3},
      number       = {5},
      issn         = {2575-1077},
      address      = {Heidelberg},
      publisher    = {EMBO Press},
      reportid     = {FZJ-2021-01208},
      pages        = {e202000670 -},
      year         = {2020},
      note         = {Rheinisch-Westfälische Technische Hochschule Aachen
                      UniversityBundesministerium für Bildung und Forschung
                      (BMBF) grant 031A536.},
      abstract     = {The common foodstuff garlic produces the potent antibiotic
                      defense substance allicin after tissue damage. Allicin is a
                      redox toxin that oxidizes glutathione and cellular proteins
                      and makes garlic a highly hostile environment for
                      non-adapted microbes. Genomic clones from a highly
                      allicin-resistant Pseudomonas fluorescens (PfAR-1), which
                      was isolated from garlic, conferred allicin resistance to
                      Pseudomonas syringae and even to Escherichia coli.
                      Resistance-conferring genes had redox-related functions and
                      were on core fragments from three similar genomic islands
                      identified by sequencing and in silico analysis. Transposon
                      mutagenesis and overexpression analyses revealed the
                      contribution of individual candidate genes to allicin
                      resistance. Taken together, our data define a multicomponent
                      resistance mechanism against allicin in PfAR-1, achieved
                      through horizontal gene transfer.},
      cin          = {IBG-4},
      ddc          = {570},
      cid          = {I:(DE-Juel1)IBG-4-20200403},
      pnm          = {899 - ohne Topic (POF3-899)},
      pid          = {G:(DE-HGF)POF3-899},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {32234751},
      UT           = {WOS:000538076900009},
      doi          = {10.26508/lsa.202000670},
      url          = {https://juser.fz-juelich.de/record/890800},
}